Introduction: Gentamicin (GM) is a frequently used aminoglycoside for managing
serious illnesses; nonetheless, renal complications limit its use. Bee venom (BV) is
a biological toxin that exhibits anti-inflammatory and antioxidant activities. This
study was designed to explore the mitigating effect of BV remediation on GM
induced renal injury.
Methods: Twenty male rats were divided into four groups (five rats each), namely,
control (saline subcutaneously); BV group (1 mg/kg S/C twice weekly for 1
month); GM group (100 mg/kg i. p. for 1 week); and GM-BV group (the same
aforementioned dosages ofGMand BV, withGMadministered in the last week for
4 weeks).